Daniel Henry DEVANEY, III

Male 1928 - 2012  (83 years)

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  • Name Daniel Henry DEVANEY  [1, 2, 3, 4, 5
    Suffix III 
    Born 2 Aug 1928  St. Louis County, Missouri Find all individuals with events at this location  [5, 6, 7, 8
    Gender Male 
    Baptism 16 Sep 1928  Faith Lutheran Church, St. Louis, Missouri Find all individuals with events at this location  [5
    Census 1930  St. Louis (Independent City), Missouri Find all individuals with events at this location  [5, 9
    Census 1940  St Louis, ?St Louis City, ?Missouri Find all individuals with events at this location  [5, 10
    Education 1940-attending school, highest grade completed-6th; 1946-grad of Beaumont High School, St. Louis, Missouri  [5, 11, 12
    Education 1946-48 - Washington U., St. Louis, Missouri in School of Architecture  [5, 13
    Education AB Economics 1950 from Drury College (now University), Springfield, Missouri  [5, 14
    Occupation Retailing  [5, 15
    Religion Protestant  [5, 16
    _UID 950060C524A0481485C4A203C03ABB46C36E 
    Died May 2012  Honolulu, Honolulu, Hawaii Find all individuals with events at this location  [5, 17, 18
    • in rehab at Arcadia
    Buried 15 Jun 2012  National Memorial Cem. Of The Pacific, Honolulu, Hawaii Find all individuals with events at this location  [5, 19
    • ?Punchbowl?
    Person ID I2480  Clan Home Genealogy
    Last Modified 4 Dec 2015 

    Family Living 
     1. Living
    Last Modified 4 Dec 2015 
    Family ID F1228  Group Sheet  |  Family Chart

  • Event Map
    Link to Google MapsBaptism - 16 Sep 1928 - Faith Lutheran Church, St. Louis, Missouri Link to Google Earth
    Link to Google MapsDied - May 2012 - Honolulu, Honolulu, Hawaii Link to Google Earth
    Link to Google MapsBuried - 15 Jun 2012 - National Memorial Cem. Of The Pacific, Honolulu, Hawaii Link to Google Earth
     = Link to Google Earth 

  • Photos At least one living or private individual is linked to this item - Details withheld.

  • Notes 
    • !BIRTH: Born in the Christian Hospital, North St. Louis City (now closed and moved to the North County). Birth Certilticate # 9242 in possession of Dan H. Devaney III. Had tonsils removed at age seven at the De Paul Hospital on 12 July 1935. A total bill of $17.00 was paid and the statement is in the files.

      !RESIDENCE: 1928-56: 8615 Park Lane, St. Louis Missouri (with parents) 1956-58:930 Dyderdown; Ferguson, Missouri 1958-59: ? Hanley Road; University City, Missouri 1959-?1365 Zurich Drive; Florissant, Missouri Plaza Square; St. Louis, Missouri ? Broome Avenue; Atlantic Beach, New York 6 Merillon AVenue, Garden City, New York?
      Andalusia; Jacksonville, Florida 8 Haddington Court; Clayton, Missouri Clayprice; Ladue, Missouri
      13080 Anza Drive; Saratoga, California 1386 Chelsea Court; Los Altos, California3428 Lake Albano Circle; San Jose, California 1992-93:Leuanuu; Mililani, Hawaii
      1993-95: Queen Victoria; 1080 S. Beretania: Honolulu HI 1995-1350 Ala Moana Blvd. #2010; Honolulu, Hawaii

      !SSN: 494 28 7004.

      !RELIGION: Baptized Sept 16th 1928 in Faith Ev. Lutheran Church by Alfred L. Grewe Pastor. Enrolled on the Cradle Roll Sept 20th 1928. Form in possession of Dan H. Devaney III.

      !EDUCATION: Attended Peter Herzog grammer school, Beaumont High School both in St. Louis, Missouri and Drury College in Springfield Missouri. Graduated in May 1950 with an AB in Economics. Member of Kappa Alpha Frat. Attended two years at the Arch. school, Washington University, St. Louis, MO. prior to Drury College.

      !PROFESSION: First full time job was with Famous Barr Co., (May Department Stores) St. Louis Missouri. Was a Buyer. Also with Stix and Vandervorts in St. Louis. Moved to The Singer Co, was transferred to California and retired as General Manager of the Stevens Fabric Company, a division of the Singer Co.
      After Singer, spent 1 1/2 years as Operations and Personnel Manager with Home Yardage of California. After that spent at least 12 years in various voluntary positions working with disadvantaged children in Santa Clora County, California and Honolulu, Hawaii.

      !MILITARY: Service # 780 16 73. Called to active duty with the Navy reserve in 1952 during the Korean War. Had a rating of Yoeman Third Class and was attached to VF 101, a Fighter Squadron, and saw service in Florida, Cuba, Haiti, Jamaca and the Med.

      !MARRIAGE: Certificate dated 28 June 1958 under license No. 666474 in possession of Dan H. Devaney

      !NOTES: "Gramps" to Caryn Maggie Devaney.

      BIOGRAPHY: Daniel Henry Devaney III - Statement
      Born 2 August 1928 at Christian Hospital in North St. Louis, Missouri.
      (The hospital is no longer there, it moved to North St. Louis County, Missouri.
      Lived at 8615 Park Lane in North St. Louis, between Baden and Jennings.
      My parents bought the house after I was born and we moved from Athlone Ave, St.
      Louis when I was nine months old. We lived there until 1957 when we moved to 930 Dyerdown Drive in Ferguson, Missouri.
      I was an only child; my mother and dad had been married eleven years when I came upon the scene.
      I attended the Peter Hertzog elementary school in North St. Louis, (K-8) then went to Beaumont High School on Natural Bridge Blvd and graduated in June 1945.
      After high school I attended Washington University Architecture School for two years and then decided that I would not make a good architect and transfered to Drury College in Springfield, Missouri. While at Washington U.
      I joined The Kappa Alpha Order, a social fraternity which I continued at Drury.
      I graduated in May 1950 with an AB in Economics.
      This was at the time of the outbreak of the Korean War and since I was in the US Navy Reserves no one would hire me. At the end of a couple of months I was hired by Famous Barr Co., division of the May Department Stores chain, and worked until 1952 when I was called to active duty in VF 101 (a naval fighter squadron) as a Yeoman. I was discharged in Dec.
      1953 and returned to Famous Barr and worked up to the position of Buyer.
      I left Famous in 1960 and we moved to New York, N.Y. and then in about 1962 returned to May Co in Jacksonville Florida. In about 1964 we returned to St. Louis with another department store which went out of business in about two or so years. I spent a few years at Stix Baer & Fuller in St. Louis before changing to The Singer Company where I spent the next ten years working as a soft lines manager, regional soft lines manager, area soft lines manager and then as general manager of the division of Singer called Stevens Fabrics in San Jose, California. Had the dubious honor of closing the firm when Singer decided to go out of the fabric business. Moved to Home Yardage as Operations and Personnel Manager. This firm also decided to close and I found myself without a job and no chance of finding one unless we moved out of California. At this time I decided to retire.
      In retirement I became very active in volunteer work, basically with troubled youth in Juvenile Hall and the community.
      We moved to Honolulu, Hawaii in Sept 1992 and I was active in two youth programs until the apple of our eye, Caryn Maggie, was born and at that time I withdrew from the volunteer work in order to spend as much time as possible with my wonderful wife and our first grandchild.
      As to how we met, please see Marilyn's page. All I can say is that my life has been without a doubt the most satisfying, rewarding, and enjoyable possible. When my bride of 37 years agreed to be mine it opened a world of true love and care that not many men achieve. Our son is so deeply loved that he could never know until now that he has a wife and daughter of his own. [5]
    • (Medical):

      {8 March 2005 11:15 AM appointment with Dr. Hu at Kaiser. Dr. Hu advised that there was another regrowth on my right eye. He wants to do a biopsy.

      9 March 2005 11:00 AM. Procedure completed by Dr. Hu and a biopsy sent to pathology.

      16 March 2005 1:00 PM. Pathology report back and first mention of Melanoma. Sample sent to lab in Boston, Massachusetts. Report should be back in two weeks. Dr Hu will call when in. Next step of treatment to be decided based on report.}

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      Last Updated: 11/25/2001
      Also referred to as:
      Malignant Melanoma

      Melanoma is a type of cancer that originates within the cells that form pigment or melanin. These cells are called melanocytes and can be found throughout the body. While melanoma is most commonly found on the skin, it can also occur inside the eye as well as on the surface. The pigmented areas of eye such as the choroid and iris are most commonly affected; however, melanoma sometimes occurs on the conjunctiva as well.

      There appears to be a strong association with the development of melanoma and exposure to ultraviolet light. People at particular risk for developing melanoma are those who sunburn easily, have fair skin, light hair and eyes. Most people with melanoma have had excessive exposure to the sun or ultraviolet light at some point in their lives.

      Choroidal melanoma is the most common form of ocular melanoma. The choroid is a very pigmented layer that lies just behind the retina. With this type of malignancy, the chance of retaining vision in the affected eye is low, but the overall prognosis is often good. The primary concern is the risk of the cancer spreading to another area of the body. The risk is proportional to the size of the tumor, proximity to the optic nerve, visual symptoms, and whether the tumor has documented growth. Those with a tumor that is greater than 2 mm thick or is close to the optic nerve have a higher risk of the melanoma spreading or metastasizing. An individual with none of the above risk factors (e.g., small tumor situated away from the optic nerve, normal vision, and no documented growth over time) may have a very low risk of metastasis.

      When a patient is suspected of having choroidal melanoma, the ophthalmologist may order ultrasound of the eye, MRI, or CT scan in addition to an extensive evaluation. Ultrasound may be particularly helpful to distinguish melanoma from another type of mass inside the eye.

      The appropriate treatment depends largely on the size and location of the melanoma. In general, small tumors that cause no visual symptoms and are not close to the optic nerve may be carefully observed for signs of growth or change. The melanoma is measured and documented with ultrasound, photography, and dilated eye examinations. Small tumors are sometimes treated with laser photocoagulation. Medium and large choroidal melanomas are usually treated either by surgically applying radioactive plaque to the eye or by removing the eye completely (enucleation). The advantage of the plaque therapy is the amount of radiation delivered to healthy eye tissue is minimized. Other methods of treatment include charged particle irradiation and transpupillary thermotherapy (TTT). The results of charged particle irradiation are similar to radioactive plaque therapy. Transpupillary thermotherapy may be used to eradicate small and medium sized choroidal melanomas. This treatment may have fewer complications than radiation therapy or laser photocoagulation.

      The Collaborative Ocular Melanoma Study, presently ongoing at three centers in the U.S., has randomized patients with choroidal melanomas to either radioactive plaque or complete removal of the eye. The results of these studies will soon affect treatment recommendations for these patients.

      Conjunctival Melanoma

      Conjunctival malignant melanoma is a rare malignancy of the conjunctiva, the eye's outermost covering. Most conjunctival malignant melanomas arise from a condition called primary acquired melanosis (pigmentation). The malignancy may also arise from a freckle or nevus on the conjunctiva, or even healthy tissue. A conjunctival melanoma cannot be diagnosed with an eye examination. Any brown, speckled, pigmented area that changes over time is suspicious. A definitive diagnosis can only be made after surgical excision of suspicious lesions. The melanoma is usually removed using a "no-touch" technique, followed by freezing (cryotherapy) of the surgical margins. Good vision in the affected eye is often preserved.

      Like melanoma of the skin, conjunctival malignant melanoma may metastasize to the another area of the body. If this occurs, the patient's survival depends on the origin of the tumor, i.e. whether the tumor arose from primary acquired melanosis, a conjunctival nevus, or healthy tissue. In many cases, this cannot be determined. The prognosis is often good but those diagnosed with conjunctival melanoma must have a systemic evaluation and routine follow-up evaluations.

      Iris Melanoma

      Malignant melanoma of the iris is a rare type of eye cancer. The tumor is often first noticed by either the patient, family member, or a friend as a "dark spot" on the iris. This finding should always prompt an eye evaluation.

      It is often difficult to determine whether a pigmented iris lesion is malignant. The most reliable sign of malignancy is if there is documented growth over time. Most ophthalmologists use photography to document the size and characteristics of suspicious iris lesions. However, if the lesion is associated with problems such as: abnormally shaped pupil, growth into the angle of the eye, glaucoma, cataract, or spontaneous hyphema, this may influence the decision to remove the suspicious lesion. The decision is a difficult one, since it is impossible to know for sure whether the lesion is malignant until it is removed. Unfortunately, removing a tumor from the iris may result in significant ocular trauma as well as reduced vision and other complications. However, it is a viable option for most patients since a malignant lesion, if not removed, may metastasize to other areas of the body and result in eventual death.

      Melanoma of the Skin

      Malignant melanoma of the skin should be suspected in any pigmented skin lesion or mole which changes color, shape, or size. This type of cancer rarely occurs in the eyelids. If melanoma is suspected, it is usually treated with wide excision to increase the chance of completely removing the cancerous cells. Malignant melanoma of the skin may metastasize to regional lymph nodes and distant organs. Therefore, early recognition and removal are vital.

      Related Conditions

      * Cataract
      * Glaucoma
      * Hyphema

      Related Eye Procedures

      * Treatment of Eyelid Skin Cancers

      Related Specialties

      * Cataract / General
      * Cornea
      * Oculoplastic

      Related Tests

      * Angiography
      * CT-Scan
      * MRI
      * Ophthalmoscopy
      * Slit-lamp examination
      * Ultrasound examination
      * Visual acuity}


      This site's design is only visible with stylesheets turned on in a graphical browser, but its content is accessible to any browser or Internet device. (Why we do this.)
      Melanoma of the eye (Ocular melanoma)

      This section gives information about a rare type of cancer called ocular melanoma (melanoma of the eye). You may find it helpful to read it alongside CancerBACUP's section on malignant melanoma.

      * What is ocular melanoma?
      * What causes ocular melanoma?
      * Signs and symptoms
      * How is it diagnosed?
      * Treatment
      * Research trials
      * Follow-up
      * Your feelings
      * References

      What is ocular melanoma?

      Ocular melanoma is melanoma of the eye. Melanoma is a cancer that develops from cells called melanocytes. Melanocytes produce the dark-coloured pigment melanin, which is responsible for the color of our skin. These cells are found in many places in our body including the skin, hair and lining of the internal organs, including the eye.

      Most melanomas begin to grow in the skin, but it is also possible for a melanoma to begin in other parts of the body, such as the eye.

      Within the eye itself, melanoma can develop in one of several places (see diagram). The most common type of ocular melanoma is uveal melanoma. This means it occurs along the uveal tract (the dark ring on the diagram) of the eye, which includes the choroid, ciliary body and iris.
      The structure of the eye
      The structure of the eye (d)

      The choroid is part of the lining of the eyeball and is dark-coloured (pigmented) to prevent light being reflected around the inside of the eye. The ciliary body extends from the choroid and focuses the eye by changing the shape of the lens. The iris is the clearly visible coloured disc at the front of the eye, which controls the amount of light entering the eye. All these structures are heavily coloured with melanin.

      Melanoma can also occur in the thin lining over the white part of the eye (the conjunctiva) or on the eyelid, but this is very rare.

      What causes ocular melanoma?

      This is a rare type of tumour and as for many other forms of cancer the exact cause is unknown. It is known that exposure to ultraviolet (UV) rays (either from the sun or sunbeds) increases the risk of developing melanoma of the skin. People whose skin burns easily are most at risk: typically, people with fair skin, fair or red hair and blue eyes. However, it is not yet known whether there is any link between UV ray exposure and the development of melanoma of the eye.
      back to top
      Signs and symptoms

      Symptoms include blurred vision, flashing lights, shadows and misting of the lens of the eye (cataract). Often no symptoms are noticed until the tumour is quite large.

      All of these symptoms are common to other conditions of the eye, but it is generally possible for an eye specialist (ophthalmologist) to diagnose these tumours quite simply and painlessly. Occasionally a biopsy (taking a small sample of tissue) is needed to confirm a diagnosis.
      back to top
      How is it diagnosed?

      A number of tests may be done to diagnose ocular melanoma, including:

      * ophthalmoscopy A small hand-held microscope (ophthalmoscope), similar to those used by opticians during routine eye tests, is used to look at the inside of the eye. This is likely to be the first test that you have
      * ultrasound scan A small device which produces sound waves is rubbed over the skin around the eye area. The echoes are then converted into a picture by a computer
      * fluorescein angiography A dye (injected into a vein in your arm) flows through the bloodstream to the eye, where it highlights any growths or damage. X-rays are taken and the highlighted areas show up clearly on the film
      * CT (computerised tomography) scan A CT scan takes a series of x-rays to build a three-dimensional picture of the inside of the head. The scan is painless but takes 10 minutes, longer than a standard x-ray. It may be used to find the tumour within the eye or to check for any spread of the disease
      * MRI (magnetic resonance imaging) scan This type of scanner uses magnetism instead of x-rays to form a series of pictures of the inside of the head. The test can take about 30 minutes and is completely painless, although the machine is noisy and you will be given earplugs or headphones to wear
      * biopsy A small sample of tissue may be taken from the suspicious area and examined under a microscope. However, this is not necessary for most ocular melanomas because they have a distinctive appearance and can usually be recognised easily from the x-rays and scans.


      A number of different treatments are used for ocular melanoma depending on the size, cell type and position of the tumour, as well as other factors such as your general health, age and level of vision in both eyes. The aim of the treatment is to destroy the cancer cells, stop the cancer coming back, and save as much of your vision as possible.


      This type of treatment uses high-energy rays to destroy the cancer cells while doing as little harm as possible to normal cells. Radiotherapy may be given either from outside the body (external radiotherapy) or from within (internal radiotherapy). Radiotherapy may be the only treatment or it may be given after surgery. Recent developments in radiotherapy have made it possible to preserve sight in the eye, either completely or partly.

      External radiotherapy

      In external radiotherapy a beam of radiation is directed to the area of the tumour. The treatment is normally given as small doses, called fractions, over a few days. Different types of radiotherapy machines can be used. One, called a Cyclotron, is specifically used to treat eye tumours. This machine directs a proton radiation beam precisely at the affected area, causing as little radiation exposure as possible to the surrounding healthy eye tissue. Before the treatment, a minor operation is carried out to attach small tags to various parts of the eye. The tags act as markers for the radiation beam.

      Internal radiotherapy

      This treatment is given by placing a radioactive source (called a plaque) close to the tumour. This normally involves a stay in hospital of up to a week. The radioactive plaque is placed close to the tumour in the eye under general anaesthetic. Another operation is carried out to remove it when the treatment is finished. Certain precautions need to be taken while the plaque is in your eye. You will need to stay in one room and each member of staff and your visitors will only be allowed in for a short time each day. This is to reduce any unnecessary exposure to radiation. Once the radioactive source is removed no precautions will be necessary, as the radiation will no longer be present.

      Transpupillary thermotherapy (TTT)

      This can be used to treat very small ocular melanomas or as an additional treatment after radiotherapy. The tumour is heated with a special type of laser beam. Cancer cells are more susceptible to heat than normal cells and so will be destroyed. Several treatments are normally needed.


      Depending on the size and position of the tumour, it may be possible to remove it without needing to remove the eye. However, if the cancer is growing rapidly, is large or painful, removal of the eyeball may be the most appropriate treatment. This is called enucleation. For many people, this suggestion can be quite shocking and a lot of discussion may be needed with the doctors involved before the decision to go ahead is taken. You can have an artificial eye (prosthesis) made that matches your remaining eye. An implant can be inserted which makes the artificial eye move realistically.

      Research trials

      Research into treatments for ocular melanoma is ongoing and advances are being made. Cancer doctors use clinical trials to assess new treatments. Before any trial is allowed to take place an ethics committee must have approved it and agreed that the trial is in the interest of patients.

      You may be asked to take part in a clinical trial. Your doctor must discuss the treatment with you so that you have a full understanding of the trial and what it means to take part. You may decide not to take part or to withdraw from a trial at any stage. You will then receive the best standard treatment available.

      CancerBACUP has a section that explains how clinical trials are set up and answers common questions that people have about them.


      After your treatment is completed, you will have regular check-ups and possibly scans or x-rays. These will probably continue for several years. If you have any problems, or notice any new symptoms between these times, let your doctor know as soon as possible.

      For people whose treatment is over apart from regular check-ups, CancerBACUP's section on adjusting to life after cancer treatment gives useful advice on how to keep healthy and adjust to life after cancer.

      Your feelings

      You may experience many different emotions including anger, resentment, guilt, anxiety and fear. These are all normal reactions and are part of the process many people go through in trying to come to terms with their illness.

      CancerBACUP has a section that discusses the emotional effects of cancer and how to deal with them.


      This section has been compiled using information from a number of reliable sources including;

      * Oxford Textbook of Oncology (2nd edition). Souhami et al. Oxford University Press, 2002.
      * Cancer and its Management (4th edition). Souhami and Tobias. Oxford Blackwell Scientific Publications, 2003.
      * The Textbook of Uncommon Cancers (2nd edition). Raghavan et al. Wiley, 1999.

      For further references, please see the general bibliography.

      Content last reviewed: 01 March 2004
      Page last modified: 26 August 2004

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      29 March 2005-Call from Dr. Hu. Boston confirms melanoma. Slow developing cells. Dr. Hu wants to watch for any future development. Joy is to call to set up mid April appointment. Probably every month in the future. Dr. Hu will remove any cells that he finds and will be more aggressive in the removal. I am to keep out of the sun as much as possible and to wear sun glasses. Nothing much more I can do to help at this point in time.

      29 March 2005-Joy called, appointment set up for 12 April 2005 at 1:00 PM

      30 March 2005- Dr Hu called at 5:30 PM. He had a meeting with his colleagues and also talked with Dr. O'Connor. The group wants to set up an appointment with a Oncologist. Joy will called tomorrow to set up an earlier appointment with Dr. Hu so he can chart my right eye to provide the Oncologist with a better road map. Dr. Hu will contact the Oncologist to start the process. That office will call to set up an appointment.

      5 April 2005 11:00 AM. Met with Dr. Hu. He could not see any spreading to the eye lid and/or outside area. I have an appointment with Dr. Mueh at the hospital on Wed. April 13, 2005 at 4:00 PM. Dr. Hu is also trying to reach a specialist in Kaiser Oakland, Dr. Gritts. I may have to make a trip to Oakland sometime in the future. Dr. Hu will be off island that week but will return on the 19th and contact me as soon as he has any information.

      April 13, 2005-Marilyn and I met Dr. Mueh. Dr Mueh will set up a PET scan at Queens and I will hear from him as to the date and a follow up appointment. This is to see if there has been any spreading of the cancer. He does not expect any. Dr. Mueh does not seem to believe in Chemo. Dr Mueh's nurse, Mary Jo, checked my weight and height as 183 lbs and 5 foot 9 and 1/4 inches.

      Apr 14. Had a call from Josephine at Queens Hospital. She set up a PET scan for tomorrow, Apr. 15 at 12:30 PM. I called Mary Jo at Dr. Mueh's office. She wasn't there but another woman set up an appointment for me with Dr. Mueh on Apr 21 at 2:30 PM. (this was cancelled as it was unnecessary, see below)

      Apr 18, 2005. had a call from Dr. Mueh. My PET scan came back as normal which means that the cancer has not spread to other parts of my body. I am waiting for a call from Dr. Hu around Wed. Apr 20th after he returns from a meeting in Washington.

      May 2, 2005. I have an appointment at 10:30 AM for another procedure with Dr. Hu. It will be somewhat more extensive than those in the past.

      Above procedure was completed and seemed to go OK. Have a follow up appointment on:

      10 May 2005, 9:30 AM. Dr Hu found another group of cells in the 11:00 position. He feels that he was concentrating on other areas and just missed these. Dr Hu does not feel that these are new growths but of course, they may be.

      I have another appointment on Thursday, May 12th to remove the above cells.

      May 12, 2005 at 3:15 PM. Procedure.

      May 19th at 11:30 AM. To check results. Dr Hu wants to set up an appointment with a Dr. at Queens on May 27th, 2005 at 8:30 AM with Dr. Thanh Van Huynh. Sounds like "Win".

      Must bring a list of meds.

      27 May 2005. Had a consult with Dr. Thanh V. Huynh with Pacific Radiation Oncology, LLC at Queens Medical Center. Dr. Huynh will check with peers here and on the mainland and then contact with Dr. Hu at Kaiser. It seems he will recommend starting as soon as possible on a treatment plan of special drops in my right eye and possibly radiation seeds to the area of problem. I need to ask the Drs what is the time factor as well as what I can expect if the treatments do not prove successful. I do know that I may loose my eye in the future but I do not know how long I have and what medical problems I will face.

      I am to call Dr. Hu midweek June 1 or June 2 to ask where I stand. I have an appointment with Dr. Hu for June 9th.

      9 June 2005, Saw Dr. Hu. He found a few little cells and removed them. At this point in time he and Dr Huynh feel that any treatment with radiation would cause more possible damage than good. He wants to wait and see if my problem can be taken care of with surgery.

      I have a follow up appointment for 10:30 Fri. July 8, 2005.

      Dr Hu removed two small areas which he removed. The last biopsy was the same as before.

      Next appointment is Aug. 8th at 11:30 AM.

      Aug. 8, 2005. Eye was clear.

      Next appointment is Sept. 13 2005 at 10:30 AM. Had to cancel because of back problems.

      Next, Oct 27 10:45
      Had two small areas, set up procedure for

      Nov. 3, 2005 3:45 PM. Had procedure. tested positive - next:

      Dec. 8 2005 10:15 AM. Follow up

      Dec. 8th 2005, small area,
      Appointment made for
      Monday Dec. 12th 2005 at 3:30 PM. with Dr Hu.
      Had procedure.

      Jan 2006, No procedure necessary

      Feb 15 1:15 PM had procedure with Dr Hu

      March 15 1:15 PM Dr Hu

      On Feb. 16 2006 had appointment with Dr Mita. He shaved a small area on left arm. Dr Mita called Tuesday Feb 21st with the news that I have a Squamous cell carcinomas on my left arm. He made an appointment for me to see

      March 2, 2006 at 11:00 AM.
      Dr. Paul Glen at the clinic for surgical removable on Thursday,

      2 Mar 2006, Had prodecure with Mr. Glen, Squamous was removed

      Thurday March 16th 2006
      Follow up to remove 7 stitches at 11:00 AM

      "Q. What do squamous cell carcinomas look like?

      A. Squamous cell carcinomas are most common on the limbs, head and neck. They are pink and irregular in shape, usually with a hard, scaly or horny surface, although they can sometimes become an ulcer. The edges are sometimes raised. They can be tender to the touch."

      The mission of the Foundation is to decrease the incidence of skin cancer through public and professional education, medical training, and research.




      About Squamous Cell
      Squamous Cell

      Squamous cell carcinoma (SCC), the second most common skin cancer after basal cell carcinoma, afflicts more than 200,000 Americans each year. It arises from the epidermis and resembles the squamous cells that comprise most of the upper layers of skin. SCCs may occur on all areas of the body including the mucous membranes, but are most common in areas exposed to the sun.

      Although SCCs usually remain confined to the epidermis for some time, they eventually penetrate the underlying tissues if not treated. When this happens, they can be disfiguring. In a small percentage of cases, they spread (metastasize) to distant tissues and organs and can become fatal. SCCs that metastasize most often arise on sites of chronic inflammatory skin conditions or on the mucous membranes or lips.
      What Causes It

      Chronic exposure to sunlight causes most cases of squamous cell carcinoma. That is why tumors appear most frequently on sun-exposed parts of the body: the face, neck, bald scalp, hands, shoulders, arms, and back. The rim of the ear and the lower lip are especially vulnerable to the development of these cancers.

      Squamous cell carcinomas may also occur where skin has suffered certain kinds of injury: burns, scars, long-standing sores, sites previously exposed to X-rays or certain chemicals (such as arsenic and petroleum by-products). In addition, chronic skin inflammation or medical conditions that suppress the immune system over an extended period of time may encourage development of squamous cell carcinoma.

      Occasionally, squamous cell carcinoma arises spontaneously on what appears to be normal, healthy, undamaged skin. Some researchers believe that a tendency to develop this cancer may be inherited.

      Who Gets It

      Anyone with a substantial history of sun exposure can develop squamous cell carcinoma. But people who have fair skin, light hair, and blue, green, or gray eyes are at highest risk. Those whose occupations require long hours outdoors or who spend extensive leisure time in the sun are in particular jeopardy. Dark-skinned individuals of African descent are far less likely than fair-skinned individuals to develop skin cancer.

      More than two thirds of the skin cancers that individuals of African descent develop are SCCs, usually arising on the sites of preexisting inflammatory skin conditions or burn injuries. Although dark-skinned individuals of any background are less likely than fair-skinned individuals to develop skin cancer, it is still essential for them to practice sun protection.

      Precancerous Conditions

      Certain precursor conditions, some of which result from extensive sun damage, are worth noting. They are sometimes associated with the later development of SCC. They include:

      Actinic, or solar, keratosis
      Actinic keratoses are rough, scaly, slightly raised growths that range in color from brown to red and may be up to one inch in diameter. They appear most often in older people. Some experts believe that actinic keratosis is the earliest form of SCC.

      Actinic cheilitis is a type of actinic keratosis occurring on the lips. It causes them to become dry, cracked, scaly, and pale or white. It mainly affects the lower lip, which typically receives more sun exposure than the upper lip.

      These white patches or plaques on the tongue or inside of the mouth, arising in the mucous membranes, have the potential to develop into SCC. They are caused by sources of chronic irritation, including smoking or other tobacco use, and rough teeth or rough edges on dentures and fillings. Leukoplakia on the lips are mainly caused by sun damage.

      Bowen?s disease
      This is generally considered to be a superficial SCC that has not yet spread. It appears as a persistent red?brown, scaly patch which may resemble psoriasis or eczema. If untreated, it may invade deeper structures.

      Regardless of appearance, any change in a preexisting skin growth, or the development of a new growth or open sore that fails to heal, should prompt an immediate visit to a physician. If it is a precursor condition, early treatment will prevent it from developing into SCC. Often, all that is needed is a simple surgical procedure or application of a topical chemotherapeutic agent.

      Squamous cell carcinomas occur most frequently on areas of the body that have been exposed to the sun for prolonged periods. Usually, the skin in these areas reveals telltale signs of sun damage, such as wrinkling, changes in pigmentation, and loss of elasticity.
      Warning Signs of Squamous Cell Carcinoma

      A wart-like growth that crusts and occasionally bleeds.

      A persistent, scaly red patch with irregular borders that sometimes crusts or bleeds.

      An open sore that bleeds and crusts and persists for weeks.

      An elevated growth with a central depression that occasionally bleeds. A growth of this type may rapidly increase in size.

      A persistent, scaly red patch with irregular borders that sometimes crusts or bleeds.

      An open sore that bleeds and crusts and persists for weeks.
      Treatment Options

      After a physician?s examination, a biopsy will be performed to confirm the diagnosis of SCC. This involves removing a piece of the affected tissue and examining it under a microscope. If tumor cells are present, treatment (usually surgery) is required.

      Fortunately, there are several effective ways to eradicate SCC. The choice of treatment is based on the type, size, location, and depth of penetration of the tumor, as well as the patient?s age and general state of health.

      Treatment can almost always be performed on an outpatient basis in a physician?s office or at a clinic. A local anesthetic is used during most procedures. Pain or discomfort is usually minimal with most techniques, and there is rarely much pain afterwards.

      Curettage and Electrodesiccation

      As with AKs, the growth is scraped off with a curette and the tumor site desiccated with an electrocautery needle. But when treating BCCs or SCCs, the procedure is typically repeated a few times to help assure that all cancer cells are eliminated. Local anesthesia is required.

      Excisional Surgery

      Along with the above procedure, this is one of the most common treatments for BCCs and SCCs. Using a scalpel, the physician removes the entire growth along with a surrounding border of apparently normal skin as a safety margin. The incision is closed, and the growth is sent to the laboratory to verify that all cancerous cells have been removed.


      X-ray beams are directed at the tumor. Total destruction usually requires several treatments a week for a few weeks. This is ideal for tumors that are hard to manage surgically and for elderly patients who are in poor health.

      Mohs Micrographic Surgery

      The physician removes the visible tumor with a curette or scalpel and then removes very thin layers of the remaining surrounding skin one layer at a time. Each layer is checked
      under a microscope, and the procedure is repeated until the last layer viewed is cancer-free. This technique has the highest cure rate and can save the greatest amount of healthy tissue. It is often used for tumors that have recurred or are in hard-to-treat places such as the head, neck, hands, and feet.


      This is the most widely used treatment for individual AKs. It is especially useful when a limited number of lesions are present. Liquid nitrogen is applied to the growths with a cotton-tipped applicator or spray device. This freezes them without requiring any cutting or anesthesia. They subsequently blister or become crusted and fall off. Some temporary redness and swelling can occur. In some patients, pigment may be lost.

      Laser Surgery

      The skin?s outer layer and variable amounts of deeper skin are removed using a carbon dioxide or erbium YAG laser. Lasers are effective for removing actinic cheilitis from the lips and AKs from the face and scalp. They give the physician good control over the depth of tissue removed, much like chemical peels. Lasers are also used as a secondary therapy when topical medications or other techniques are unsuccessful. However, local anesthesia may be required. The risks of scarring and pigment loss are slightly greater than with other techniques.

      Photodynamic Therapy (PDT)

      PDT can be especially useful for lesions on the face and scalp. Topical 5-aminolevulinic acid (5-ALA) is applied to the lesions at the physician?s office. As soon as an hour later, those medicated areas can be activated by a strong light. This treatment selectively destroys AKs while causing minimal damage to surrounding normal tissue. Some redness and swelling can result from this newer therapy.

      16 March 2006,
      No procedure was necessary on 15 March, 2006.

      Next appointment with Dr Hu is Friday 21 April 11:30 AM.
      Friday, April 21, 2006 11:30 AM Dr Hu found a few small specks of pigment.
      Dr Hu removed specks at 4:15 PM.

      Next appointment May 30, 2006 at 11:30 AM
      All OK.

      Next June 30 at 11:00 AM.
      No problem this month.

      Set up July 25, 2006 for next check up.
      July 25th. No problems today,

      next will be in Sept.
      (Dr. Hu and wife are having baby).

      Next appointment changed to 28 Aug 2006,11:00 AM
      No procedure necessary.

      Next appointment is Friday October 6 at 11:15 AM
      No procedure necessary but Dr Hu saw a few small blood vessels that he wants to watch

      Next appointment on 17 Nov 2006, 11:30 AM. No problem.

      Next appointment on Dec 18, Monday at 11:30 AM

      No problem, next appointment is 22 Jan 2007}

      Dr Hu found specks - next procedure is 2 Feb 2007 11:00 AM.

      Next appointment March 14, 2:45 PM. No problem

      Next appointment 30 April 2007 at 11:00 AM

      Small problem, procedure set for Tues. May 8th at 1:00 PM.}

      Next appointment for check up Friday June 8th right after seeing Dr. O'Conner at 1:20 pm.

      20 July 2007-1:00 pm No problem

      23 Aug 2007-1:15 pm no problem

      Next - Sept 24 2007 10:45am Dr Hu found a bit of pigment. Set up procedure for Tues 25 Sept 2007

      25 Sep 2007- had procedure, next appointment:

      The lab work on the procedure of the 25th of Sep showed positive which ment that Dr Hu did not get all of the growth. Have next appointment on 2 Oct. to try and remove all of the tissue. Don?t know if the 11 Nov appointment still stand but don?t think so.

      2 Oct 2007 3 PM (could not make this appointment because of car crash)

      Re-set for 9 Oct 2007 at 1:30 p m
      Had procedure, Dr Hu had to go deeper into eye as lab found bad cells at outer edge of last sample on 25 Sept 2007.

      15 Oct 2007-had call from Dr Hu with good news. Test was negative.

      Next appointment
      16 Oct 2007 at 1:30 p m
      No problem

      11 Nov 2007 10:45 am
      no problem

      7 Dec 2007 11:30 am. Dr Hu found two small specks. He will remove on
      17 Dec. 10:00 AM.

      17 Dec. 2007. Procedure went OK.

      8 Jan 2008 is next appointment at 1:00 pm} Things looked but Dr. Hu wants to start treatment with an eye drop 4 times per day. It should kill the bad cells but will also kill some good ones. He thought that there were a few spots under the surface that would not be exposed to the eye drops so he set up another procedure for 15 Jan 2008 to remove the surface area.

      15 Jan 2008, Dr Hu removed the surface area and set up an appointment on 31 Jan to check it out and if the area seems OK, he will start the eye drops. We will have to pick up 2 weeks supply from Poly Momi and return the empty package for hazard waste disposal. If things OK with the eye, another 2 weeks supply is given. The treatment is 4 times per day.

      25 Jan 2008-sent Dr Hu the following message:

      ?Good Morning Dr Hu,

      Will I have an adverse reaction with the planned eye drops if I also have the new shingles vaccine?? Dr Hu advised that it would be ok to take the shot but I decided not to.

      31 Jan 2008, Dr Hu again found questionable specks in my right eye. I pick up the eye drops on 1 Feb 2008 and start 4 per day for a total of 30 days. This is to kill ?C? cells in a larger area.

      Have appointment to see Dr. Hu on 8 Feb 2008 to check on the progress on the eye drop treatment.

      8 Feb 2008, new med is starting to work. Next is 28 Feb 2008.

      28 Feb 2008-Finish 4 weeks of new treatment tomorrow. Next is 31 Mar 2008 to see if all cells were destroyed. Dr Hu thought he saw a bit of pigment, it may not be Malignant and not be destroyed, if it is still there, he will remove.

      31 Mar 2008. Follow up. The two bits of pigment were still in the eye so Dr Hu had Joy set up an appointment for 4 PM same day 31 Feb. 2008 to remove.

      31 Mar 2008 4 PM. Dr Hu removed the two bits of pigment and then scrapped some loose cells. Next appointment is April 8th at 10:45 AM.

      8 Apr 2008, Eye looked clear but one of the samples taken on the 31st may show a trace. Next appointment 29 Apr 2008 1:20 PM

      29 Apr 2008-Dr Hu found that one small area may still have pigment and set up a new appointment to check for 14 May at 1:15.

      14 May 2008 had a procedure, Dr Hu will call me and will set next time. Set for 6 June

      6 June, all clear - next is 8 July 1:00 PM

      8 July, still clear - next is Aug 12th at 1:20 pm

      August 12 2008. Right eye still clear. Left eye had a film which Dr Hu cleared up with a laser. Have to go back 8-26 the check left eye.

      12 Aug 2008, left eye OK. Dr Hu opened the two tear drains in right eye. Next appointment Friday 19 Sep 2008 - MISSED APPOINTMENT reset to 26 Sep

      26 Sep 2008, 1:05 - bad news it?s back. Set up procedure for Tues. Sep 30 8:30 am.
      Think this one is bad, just a feeling. Dr Hu will also remove some type of growth on eye lid.

      30 Sep 2008, I forgot and I missed it. Joy called and set up 1 Oct 2008

      1 Oct 2008, all OK next is 19 Nov 2008

      19 Nov 2008 - next is 9 Dec 2008

      9 Dec 2008, Dr Hu removed several XXXXX from right eye lid. Have to return on Dec. 22 2008 at 1:15 PM

      22 Dec 2008, bits of pigment, not sure if they are growing or not, recheck 19 Jan 2009

      19 Jan 2009- Had a procedure, freeze only.

      Next appointment - Tues 22 Feb. 2009 at 1:40.

      22 Feb 2009- First good news, last lab tests showed no cancer cells just pigment.

      Next appointment 31 Mar 2009, Dr Hu will remove any specks of pigment showing and then we will talk about next step.

      31 Mar 2009. Dr Hu removed a small area and set up a Cat Scan for me on May 11 2009 at the Hospital.

      11 May 2009 called and said that the Cat Scan was good and he would see me Fri. 15 May 2009 and talk with us about the eye area.

      15 May 2009. Dr Hu went over the file and listed opitions. They still don?t know where the specks of pigment are coming from and since the last lab report was negitive he will continue to watch. Nest date is June 12th at 11:30 am.

      12 June 2009-Dr Hu advised that he felt that the pigment area has not grown. Will continue to wait and watch.

      21 Jul 2009 1:00 PM. No growth in pigment area. Will continue to wait and watch.

      18 Aug 2009 1 PM. No growth in pigment area. Will continue to wait and watch.

      Next appointment 23 Sep 2009 at 11:30. Changed to Sep 29 because of flu shots.

      29 Sep 2009. All OK. Next is Nov 24.

      24 Nov. 2009-Just the same bits of pigment found. Everything looks good. Dr Hu said that I now could have cataract surgery on my right eye. There would not be any conflict.. Joy will call to set up a time when she returns from a few days off. I asked for Jan 2010 or later. No date for regular check ups set as yet.

      Joy called and I am set up for pre-op Dec 8, 1:20 pm

      The operation is on Thursday Dec 10 at the hospital 8:00 am

      Post op is 9:30 am at clinic.

      Everything worked out OK. Next reg. appointmemnt to see Dr Hu is Jan 12th 2010.

      12 Jan 2010, Everything is fine. Next is Feb 16th 2010.

      16 Feb. 2010, Nothing has changed. Next time to see Dr. Hu is 16 Mar 2010 at 1:00 pm.

      16 March 2010. Everything is good, now moving to a every other month appointment. Next is 18 May 2010. 1:00 pm

      1 May 2010- Had a third TIA about 5:30 pm. Working at computer and felt that I didn?t know how to set up and Delete a fact or event. Then felt a tightness in my chest and a light head ache. Passed in about two minutes.
      During the period took blood pressure of 139/91 77. My morning report was 123/84 85 wt. 176.5.

      18 May 2010. Saw Dr Hu, eye is fine

      20 July 2010, Dr Hu rechecked eye, still OK

      24 Sept 2010. next appointment

      24 September 2010, all still OK. Next in two months

      22 Nov. 2010- 2:15 pm all OK

      21 Jan 2011-1:30 pm all OK next two months card coming

      1 April 2011-all OK

      27 June 2011 all OK

      30 Aug 2011 all OK

      29 November 2011 all OK

      Temporal arteritis
      13 Jan 2012-Have had a headache for several weeks. Saw Dr O?Connor who ordered blood work and MRI.
      18 Jan. 2012 check for Arteritis - temporal; Cranial arteritis; Giant cell arteritis check done by Dr Hu. Also MRI preformed.

      19 Jan 2012-Dr O ?Connor reported MRI OK
      19 Jan 2012-Dr. Hu reported no problem with biopsy

      5 March 2012 - Dr Hu. All OK, The pigment is not there. Next time is in four months.

      March 2012 - My health has been a bit of a problem since Jan. It started with head aches which ended up with 6 Acetaminophen a day along with an ice pack. They had me take an MRI of the brain and brain stem (no problems there), blood work which showed me triple what I should have been for what they call The sedimentation rate (sed rate) blood test measures how quickly red blood cells (erythrocytes) settle in a test tube in one hour. The more red cells that fall to the bottom of the test tube in one hour, the higher the sed rate as well as triple the C-reactive protein rate or something like that. They put me on 60 mg prednisone right away and set me up for a Temporal biopsy the next day. It turned out negative so they stopped the prednisone right away. The drug is very powerful as well as being a ?happy pill?. They then ran a bunch of other blood tests to see if they could find where the infection was coming from. Nothing showed. What things showed was that I had an infection somewhere in the blood artery or vein system but no spot. During Feb and March they were watching and I was down to 2 Acetaminophen but still had pain in my neck, back, and head. In late March things changed a bit and I could no longer eat French Bread, (too much chewing) and then developed pain in the base of my tongue. Again, back to the head, this time on the other side of the temple with another temporal biopsy. This time, it showed what I think they call ?Temporal arteritis? which meant that I could have had a giant red cell move into the eye and cause blindness. Temporal arteritis is inflammation and damage to blood vessels that supply the head area, particularly the large or medium arteries that branch from the neck. Also called Polymyalgia rheumatica or giant-cell arteritis.. They had started the prednisone right away before the biopsy but the SED rate had doubled, normal is 0 to 15, (an older person could be maybe 18. I was 33 in Jan. and 55 in March) The C reactive protein was up also. Now they have a diagnoses so things are heading in the right direction. [5]

  • Sources 
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    5. [S835] GEDCOM from Marilyn Patterson Devaney, Marilyn Patterson Devaney, (Obtained on 18 Aug 2015).

    6. [S856] Source, 2 Aug 1928 (Reliability: 0).

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    14. [S855] Source, AB Economics 1950 from Drury College (now University), Springfield, Missouri (Reliability: 0).

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    19. [S859] Source, 15 Jun 2012, National Cem. of the Pacific, Honolulu, Hawaii (Reliability: 0).